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GO enrichment analysis showed that the upregulated DEGs were mainly enriched in apoptosis, inflammation, and oxidative stress, while the downregulated DEGs were primarily involved in gap junctions, cell proliferation, and sex hormone response (Figure 3B), suggesting that the damage to granulosa cell function, including increased apoptosis and decreased proliferation ability, may be a critical cause of follicular atresia and oocyte maturation disorders

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Changes in bloodbrain barrier permeability to large and small molecules following traumatic brain injury in mice

The results showed that 2,4-D did not exhibit any agonist activity toward human estrogenic (hER) and androgenic (hAR) receptors across the entire range of tested concentrations (Figs

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